81 YOM with Chest Heaviness

It’s approximately 2000 hrs, right as you get comfortable in bed, when you are dispatched to a residence for Chest Pain (CP). You arrive on scene to find an 81 year old male, semifowler’s in bed, complaining of chest heaviness, 8/10, which started 2 hours ago, while in bed, watching tv. The patient also advised he has vomited twice since he called 911 less than ten minutes ago.

He is alert and oriented to person, place, time and event, GCS of 15, denies dyspnea with clear bilateral lung sounds, strong and regular radial pulses, warm to touch, diaphoretic and normal skin color.

Medical history:

  • Hypertension (HTN)
  • MI
  • Hyperlipidemia

Medications:

  • ASA
  • Metoprolol
  • Plavix
  • Pravachol

Vital sings:

  • BP: 160/87 mmHg
  • HR: 96 beats/min
  • RR: 18 breaths/min
  • SPo2: 94 RA
  • BGL: 104 mg/dL

You administer O2 at 2lpm via nasal canula and placed him on your cardiac monitor, then obtain the 12 lead ECG shown below:

subendo1edit

  • What is your ECG interpretation?
  • What would be your treatment?
  • Any concerns?

4 Comments

  • Doug says:

    Sinus tach (approx 100bpm) with 1st degree block
    ST depression in leads I, AVL, II with significant ST depression in precordials V2-V6
    Right axis deviation with poor R wake progression.
    Suspect a posterior MI, transmit ECG, obtain IV access, administer 162 mg ASA, nitro, antiemetic ( IV gravol), narcotic pain control ( morphine currently). On route posterior ecg V7-V9 with second transmit, serial ecg’s, vials q 5 mins transporting to a PCI facility. Prep for a potential arrest and dysrhythmias (VT).

  • Eric L says:

    With the diffuse ST depressions significant enough to cause ST elevation in aVR, significant L main disease or 3 vessel disease are likely, and could indicate CABG rather than PCI. In a plavix naive patient, I would consider holding off on loading plavix since the patient might need CABG. I’m not sure what I would do in the case of this patient, who is already on plavix.

  • Alex says:

    1st degree AV block.

    We’ve got ST elevation in the right facing leads (maximal in aVR, some in V1) and ST depression in the left facing leads (I, II, V2-V6) making me think severe 3 vessel disease or proximal LAD or LMCA occlusion but I get very unsure at this point!

    Whichever, it’s not a good ECG to have.

    I would discuss this with the cath lab because I’m unsure how these patients are managed. Do they benefit from bypassing local hospitals to get to PPCI or would they normally be assessed in a local resus room?

  • Reni says:

    Sinus rythm,RBBB,LAFB,ST-depressions I,AVL,V2-V6

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EMS 12-Lead

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Comments
Ivan Rios
The role of 12 lead ECG in Pediatric Pulmonary Hypertension
Thanks for writing Tyler. They are the same thing. Strain pattern is just the result of increased pressures against the ventricles which alters the way repolarization occurs from epicardium to endocardium. Similar to stepping on a puddle of water. Your show spreads the water away from the area of pressure. The ST segment is slightly…
2014-12-17 18:44:24
Tyler
The role of 12 lead ECG in Pediatric Pulmonary Hypertension
Can you explain how these ST segment and T wave changes can be differentiated from right strain pattern?
2014-12-17 18:18:25
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